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Knockdown of PRDM16 in brown fat cells induces skeletal myogenesis. To examine in detail the cellular and molecular consequence of PRDM16 depletion, we used
um. Who knows what happened. Excuses happened. A bunch of them. I posted last about the # 16 that I fought for. And then, I lost all momentum. It wasn’t even a slow
Abstract. Our aim was to critically evaluate the relations among smoking, body weight, body fat distribution, and insulin resistance as reported in the
Abstract. Public health recommendations for the US population in 1977 were to reduce fat intake to as low as 30% of calories to lower the incidence of
Background Obesity results from an imbalance between energy intake and expenditure. In rodents and newborn humans, brown adipose tissue helps regulate energy
Liver and serum metabolites of obese and lean mice fed on high fat or normal diets were analyzed using ultrformance liquid chromatography−quadrupole-time-of
Trials comparing the effectiveness and safety of weight-loss diets are frequently limited by short follow-up times and high dropout rates. In this 2-year trial, we
Evidence that supports the prescription of low-carbohydrate high-fat diets: a narrative review
Next, we took a systematic approach to determine what fraction of the PRDM16-regulated genes requires C/EBP-β at the initiating step of the myoblast to brown fat
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